Experimental regimen targeting the ependyma slows disease progression in four patitents with ALS

 

Citation: Payne AG. ,’Experimental regimen targeting the ependyma slows disease progression in four patitents with ALS,’ Med Hypotheses (2009), doi:10.1016/j.mehy.2008.12.032 

Experimental regimen targeting the ependyma slows disease progression in four patients with amyotrophic lateral sclerosis

Dr. Anthony G. Payne*

 

E-mail: DrAGPayne@yahoo.com

 

SUMMARY

 

 In this paper the author proposes that at least some forms of sporadic ALS (amyotrophic lateral sclerosis) arise due to the effects of neurotoxic compounds synthesized by defective ependymal cells in the brain. These cells produce cerebrospinal fluid (CSF) that is laden with neurotoxic compounds that bring about motor neuron die-off. Evidence is garnered from various animal studies to demonstrate the toxicity of CSF taken from ALS patients and by virtue of the proposed mechanism (defective ependymal cells). In addition, a regimen created by the author is introduced; a regimen that has been used by four (4) sporadic ALS patients since 2005 resulting in what appears to be a slowing of disease progression. All four patients have significantly outlived best estimates of their survival tendered by their neurologists. 2009 Published by Elsevier Ltd.

Advertisements

About Dr. Anthony G. Payne

I am a word & ideas weaver, hypothesis spinner, artist, Celtic Catholic monk, long time member of MENSA, and a BIA certified American Indian and tribal member of the Choctaw Nation of Oklahoma. You can readily reach me by shooting an e-mail to nativescienceguy@gmail.com.

Posted on May 21, 2009, in Amyotrophic Lateral Sclerosis (ALS) and tagged , , , , , , , , , , , . Bookmark the permalink. Comments Off on Experimental regimen targeting the ependyma slows disease progression in four patitents with ALS.

Comments are closed.

%d bloggers like this: